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Research showed that animals fed a regular diet with sufficient fat-soluble antioxidants to prevent a robust increase of LPO, therefore LPO was not the relevant injury mechanism of APAP-induced liver injury in quantity, in addition, LPO was the cause of the injury rather than the outcome [73, 7780]

However, several proposed actions are believed to underlie its physiological effects: 1

Such instabilities may compromise the integrity of the vesicular structure, resulting in premature drug leakage and uncontrolled release kinetics

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The most obvious one is that the decrease can cause liver damage, resulting in more and more toxins remaining in the body, thereby causing liver damage