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Upregulation of Histone deacetylase 3 (HDAC3) in pulmonary fibrosis can lead to NRF2 inhibition, and by selectively inhibiting HDAC3 (e.g., using the drug RGFP966), the inhibitory effect on NRF2 can be reduced

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The enhanced cellular uptake results in higher intracellular concentrations of glutathione, where it can exert its antioxidant and detoxifying effects more effectively

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Hormonal optimization is often the missing piece