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The resulting SOD1 G93A /CD4 / mice did not have an altered disease onset, but had a shorter survival span and disease duration analogous to SOD1 G93A /RAG2 / mice, indicating that CD4 + T cells were responsible for the prolonged disease duration and survival in SOD1 G93A mice
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Some studies suggest the imbalanced inflammatory response results from immune system dysfunction, while others indicate a possible role for the internal microenvironment, with altered microbial composition contributing to the vulnerability of the intestinal lining to harm caused by free radical species
La supplmentation en glutathion peut ainsi aider conserver une peau clatante, tout en protgeant les organes internes contre les dommages lis l'ge
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