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[3] The proposed mechanism involves B12 altering the gene expression of skin bacteria ( Cutibacterium acnes ), leading to increased inflammatory compounds
Oncology unknowns ERR participates in some cancer-metabolism pathways, leaving long-term cancer-risk questions open
This leads to structural change in the photocobilin protein that can promote enzyme activity either through direct fusion to the enzyme domain or by protein-protein interactions
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The loss of enterohepatic recirculation would definitely cause an additional loss of 5mcg of B12 that is not recovered from bile
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