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These two signaling molecules mimic natural hormones within the body to stimulate the release of growth hormone from the pituitary gland
They come from different populations, different treatment durations, different placebo runs, and different statistical estimands (for example, the efficacy estimand that assumes continued treatment versus the treatment-regimen estimand that counts discontinuations)
Al-Karadsheh, T
metabolic differences lie in glucose processing pathways, not receptor shape
Monosodium glutamate-induced oxidative kidney damage and possible mechanisms: a mini-review
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