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(Image created with BioRender.com) Pathophysiology Hepatic steatosis in obesity results from disruption of the delicate equilibrium between hepatic fat synthesis, fat oxidation, and fat export

Nitric oxide system modulation represents a central mechanism, with BPC-157 influencing both constitutive and inducible nitric oxide synthase activity depending on tissue context and injury status

Clinical trial results: Cagrilintide weight loss data Real-world evidence from rigorous studies

You may see greater results or faster results if you also add glycolic acid to the treatment

These combined mechanisms can improve fat metabolism, reduce inflammation, and preserve lean muscle during fat loss