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Research has shown the therapeutic potential of the SGD in alleviating anti-tuberculosis drug-induced liver injury through the Nrf-2/HO-1/nf-B signaling pathway (Zhang et al., 2024)
The liver metabolises Dihexa via both Phase I oxidation (primarily CYP3A4) and Phase II conjugation (glucuronidation), converting the active compound into inactive metabolites that are renally excreted
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Strength-building activity may help preserve muscle as weight decreases
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