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A: GHK-Cu uses a very small amount of copper, keeps it tightly bound in a peptide complex that inactivates its harmful freeradical chemistry, and operates within the bodys normal copperhandling pathways, so when used as directed it is not expected to meaningfully raise systemic copper levels in people with normal copper metabolism
patients in the larsucosterol group did not receive GCs but instead received a matching placebo
Much more ethically questionable to me, is Mikes admission that he recommended BPC-157 to other people
Such a decrease was observed in serum levels of fasting insulin and HOMA-IR in the vitamin B12 group, although this change did not reach a significant level
Most patients benefit from understanding both rather than choosing one as a permanent substitute for the other
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