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Based on the aforementioned literature on ASD pathophysiology, we hypothesized that adults with ASD would show evidence of a dysregulated Gln-Glu cycle and heightened E/I balance, manifested by an increase in Gln levels, accompanied by potential alterations in Glu and lower GABA levels
Excitatory amino acid transmission: NMDA receptors and Hebb-type synaptic plasticity
Targeting nonalcoholic fatty liver disease via gut microbiome-centered therapies
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