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Mechanistically, SLC25A39 deficiency induced mitochondrial dysfunction, characterized by excessive mitochondrial reactive oxygen species (ROS), reduced membrane potential, diminished adenosine triphosphate (ATP) synthesis, aberrant mitochondrial permeability transition pore (mPTP) opening, and cytochrome c release
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Ngy th 2 n 6, tim bp mi ln 1 ngy 600mg glutathione V sinh nam gii: Tim bp trong 2 thng 600mg glutathione cch ngy
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