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High-dose, unregulated IV use, however, has been linked to liver dysfunction another reason dose and supervision matter

Taken together, these data demonstrate that loss of Chac1 expression is associated with metabolic dysregulation and fibrosis in MASLD/MASH and that restoring Chac1 expression and the resulting GSH depletion may represent a potentially novel therapeutic strategy for halting MASLD/MASH progression

Whether this newly described pathway has implication in mammals needs further verification