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It binds G-actin monomers with high affinity, sequestering monomeric actin and controlling its availability for polymerization
Elevated stress signaling may increase gastrointestinal sensitivity and amplify how nausea or digestive discomfort are perceived
These numbers show that current prevention strategies are not enough
The balance between such effects, particularly over a prolonged timeframe, including effects at other GHSR1a monomers or GHSR1a heterodimers with other receptor types, must be taken into consideration
DSIP appears to play roles in stress response, pain modulation, hormonal regulation, and withdrawal from addictive substances
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