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Obes Rev (2018) 19(2):12149
The kidney-protective effects of GLP1-RAs are believed to result from reductions in inflammation and oxidative stress, in part through direct binding to glucagon-like peptide 1 (GLP1) receptors present on kidney glomerular and tubular cells [51]
Glucagon receptor activation is minimal at 1-2mg but increases significantly at 4mg and becomes a major driver of the metabolic profile at 8mg and above
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