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The point is to ask better questions.** Could the person in front of us be living with conditions that interact in ways our traditional, single-disease pathways do not always recognise

While our previous work (12) outlined the fundamental molecular pathways of ferroptosis in eye diseases, this review advances the field by shifting the focus toward the complex bidirectional crosstalk between ferroptosis and the ocular immune microenvironment

A: Many websites sell peptides labeled "for research purposes only." That label is a legal workaround

Ca 2+ overload, which is a crucial event in eryptosis, leads to the opening of Ca 2+ -activated K + channel KCa3.1, known as the Gardos channel [90]

Compounds can leave behind metabolites or biological traces detectable for a longer period