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Available from: www.ema.europa.eu/en/news/ema-statement-ongoing-review-glp-1-receptor-agonists (cited 2025 Jun 11)

[16] The principal AOD9604 studies cited here did not establish a complete AOD9604-to-UCP1 pathway or demonstrate direct induction of UCP1 transcription, UCP1 activity, white-adipose browning, or functional thermogenesis

The primary mechanism of Semax involves significant upregulation of BDNF and nerve growth factor, which promote neuroplasticity and neuronal survival