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In contrast, when placed under metabolic stress with increased demand on the -cell (induced by high fat feeding and a low dose of the -cell toxin, streptozotocin), glucose tolerance did become impaired, supporting the view that paracrine GLP-1 may be required to maintain glucose homeostasis during adaptation to metabolic stress (114)
Postoperative WR after post-bariatric weight nadir and before adjunct semaglutide was 12.3 14.4% (mean SD)
The Journal of Clinical Psychiatry
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