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524 Additionally, Penkowa and Hidalgo demonstrated MT2 could become a prospective treatment candidate in multiple sclerosis, since it reduced cytokine expression in the CNS and prevent apoptotic neuronal death in an EAE model

doi: 10.1007/s00442-017-3968-3 187

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The peptide inhibits nuclear factor-B (NF-B) activation, a master regulator of inflammatory gene expression, while simultaneously activating peroxisome proliferator-activated receptor- (PPAR-), which promotes inflammatory resolution

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