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Due to their high rate constant and high abundance, Prx are thought to be responsible for scavenging nanomolar concentrations of H 2 O 2 associated with redox signaling, while Gpx are likely important at higher intracellular concentrations, buffering high ROS levels to avoid cell damage and stress signaling response (Sena and Chandel, 2012)

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These findings were reported in earlier studies showing that rotenone induced neural degeneration with shrunken neurons and pyknotic nuclei in rats [102]