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similarly, in the pulmonary circulation, endothelial dysfunction has been identified as an early event leading to the development of pulmonary hypertension in the context of experimental heart failure [21]

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It is possible that the high prevalence in women obeys to a uniparental dysomia or, to the high existing consanguinity or to that the chromosome X containing the normal gene is the one that inactivates itself during the genetic imprints 38

[PMID: 41317265] PubMed record related to this compound for laboratory literature review

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