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Additionally, GPxs due to their subcellular compartmentalization (cytoplasmic, membrane, ER, and mitochondrial localizations) control the local concentration gradient of peroxides and thereby regulate the activation/deactivation of redox-responsive signaling pathways [1, 8,9,10]
Such results indicate a role of oxidative stress in the pathogenesis of thyroid autoimmunity induced by exposure to excess iodine
These data provide a rationale for the evaluation of TLC-2716 to treat individuals with severe dyslipidemia and suggest that TLC-2716 may reduce residual cardiovascular risk due to benefits on both lipid and glucose metabolism
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