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HMGB1 translocation from nucleus to the cytoplasm of renal tubular cells induces ferroptosis by binding ACSL4 after renal I/R injury, and inhibition the nuclear cytoplasmic translocation of HMGB1 could inhibit ferroptosis and renal I/R injury

This activates NF-B to trigger neuroinflammation, ultimately exacerbating SNpc dopaminergic neuron apoptosis in PD (162)

BBB damage following most mild TBI in which seizures or epilepsy develop is persistent, and perivascular and distributed IgG staining results have been found in the grey matter of TBI patients who died 147 years after initial injury (111, 112)

Estrella JS, Hofstetter WL, Correa AM, Swisher SG , Ajani JA, Lee JH, Bhutani MS, Abraham SC, Rashid A, Maru DM

Oxid Med Cell Longev 2019:9613090 Szarka A, Tomasskovics B, Banhegyi G (2012) The ascorbate-glutathione-alpha-tocopherol triad in abiotic stress response