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Conclusion GLP-1 analog exendin-4 was successful in inhibiting the overexpression of cytokines associated with the induction of inflammation in CSMCs
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Unlike many peptides that require injection to avoid degradation in the digestive system, DSIP demonstrates unusual stability
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Effects of Glucagon-Like Peptide-1 Receptor Agonist Exendin-4 on the Reinstatement of Cocaine-Mediated Conditioned Place Preference in Mice
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