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Here, we show in both ex vivo and in vivo models of ischemic injury that treatment with GLP-1(2836), a neutral endopeptidasegenerated (NEP-generated) metabolite of GLP-1, was as cardioprotective as GLP-1 and was abolished by scrambling its amino acid sequence
These effects form the foundation of incretin-based obesity therapy and have been studied extensively since semaglutides approval in 2017
Chronic low-grade inflammation is now recognized as a root contributor to many modern health issues, including: Cardiovascular disease Autoimmune conditions Hormonal imbalance Metabolic dysfunction Accelerated aging GLP-1 receptors are present in immune cells, meaning GLPs can directly influence inflammatory pathways
Although exogenous ketones raise the blood concentration to the levels we wish to obtain in the initiation and maintenance of a KD, caution should be applied when comparing the two distinctly separate metabolic states of endogenous and exogenous ketosis (66)
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