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ROS may be important mediators of cellular injury during COVID-19 sepsis (Silvagno et al., 2020), either following macromolecular damage or by hindering extracellular and intracellular regulatory processes
and demonstrates anti-inflammatory properties by reducing pro-inflammatory cytokines, inhibiting NLRP3 inflammasome activation, and blocking TLR4/NF-B signaling in various pathological contexts (e.g., sepsis, hyperhomocysteinemia)
It helps improve energy levels and enhances physical performance
Wiener Medizinische Wochenschrift (2015) 165(1):39