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The remaining clusters were monocyte-derived macrophages (mono-M) that expressed Ccr5, CD33, Sell, and Itgam 34,35 (Fig

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showed that Exos from HO-1/BMSCs containing miR-29a-3p mitigated hepatic IRI by negatively regulating IREB2 protein levels, lowering intracellular Fe 2+ concentrations, increasing lipid ROS levels, and inhibiting ferroptosis [169] (Table 1)