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F.CharcossetC.FessiH
Assarsson, E
Additional analysis was performed in order to estimate the effects of cagrilintide in patients regardless of their adherence to the treatment where body weight of all patients was included and any missing data was filled in through the multiple imputation method
Porous Mater
The hyperkalemia associated with Gordon syndrome is due to the fact that WNK1 and WNK4 normally activate the renal outer medullary K + channel (ROMK, encoded by the KCNJ1 gene) present in the apical membranes of late distal convoluted tubule (DCT2) cells and connecting tubule cells
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