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A clinician can review your medical history, current dose, and risk factors to guide safer use during GLP-1 treatment

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Notably, a relative increase in the NMDAR GluN2B subunit confers slower receptor decay times enabling a greater volume of Ca 2+ influx through the receptor, thereby increasing the risk of Ca 2+ -mediated excitotoxicity and neuronal death

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patients and clinicians should refer to the primary trial publication for specific figures, as these data are dose-dependent and subject to ongoing analysis