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It also increases aquaporin-4 (AQP-4) involved in glial edema (47), and poli ADP-ribose polymerase-1 (PARP-1) that favors necrosis through glial activation (48)
Thus, studies on the combination of strategies that can penetrate a normal BBB or on the dynamics of BBB disruption, as well as on the identification of biomarkers of brain vascular injury, might pave the way to new drug delivery approaches targeting BBB in the near future
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Earlier Phase 2 data had already started the conversation as it showed a lot of promise
No representation is made regarding suitability for any application beyond in vitro laboratory research
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