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Indicatively, mitochondrial dysfunction in neutrophils enhances the formation of neutrophil extracellular traps, contributing to tissue damage and autoimmunity in SLE

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However, this does not guarantee reperfusion of the downstream capillaries supplying the myocardium 1,2 and a lack of capillary reperfusionno-reflowaffects up to 50% of patients 3,4

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