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Modifying gene expression across antioxidant, apoptotic, metabolic, and genomic stress pathways reinforces its promise as a natural therapeutic agent

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doi: 10.1146/annurev.bi.52.070183.003431 15 HaniganMHRickettsWA

Considering that the copper-associated mitochondrial toxicity may have been delayed, we further explored the changes in the expression of skin irritation-related biomarkers at the genomic level