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CDK1 directly binds and phosphorylates the S447 site of ACSL4, promoting the ubiquitination degradation of ACSL4, while genetic and pharmacological blocking of CDK1 enhances ACSL4 activity, promoting ferroptosis of colon cancer cells and increasing their susceptibility to oxaliplatin

In practical terms, the difference is small enough that standard dosing protocols do not adjust for it

Clin Exp Rheumatol 31:S22S27 Navarro-Compan V, Plasencia-Rodriguez C, de Miguel E et al (2016) Anti-TNF discontinuation and tapering strategies in patients with axial spondyloarthritis: a systematic literature review

doi: 10.1017/S0967199417000120 73 LiZ.VanceD

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