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Using C2C12 mouse myoblasts, the study confirmed that the MGF E-domain peptide increases myoblast proliferation while inhibiting terminal differentiation and critically, that this effect was maintained in the presence of a neutralising anti-IGF-1R antibody directly establishing that MGF E-peptide signals via an IGF-1R-independent mechanism

By reducing body weight, improving insulin sensitivity, lowering inflammatory markers, and addressing metabolic dysfunction, they target the very factors that make Hashimoto's management so challenging

Alemany S, Avella-Garca C, Liew Z, Garca-Esteban R, Inoue K, Cadman T, et al

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