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These drugs have anti-fibrotic characteristics by obstructing transforming growth factor- (TGF-) signaling and diminishing collagen accumulation in models of diabetic nephropathy (Tian et al
Physiology & Behavior, 241, 113588
Diabetes World 8 (10), 440446
Concentrations are therefore much higher in the fed state, and these peptides are subsequently cleaved by dipeptidyl peptidase4 (DPP-4) enzymes rendering the GLP-1 peptide inactive [26]
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