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Digoxin, used for heart failure and arrhythmias, is another narrow therapeutic window drug

[PMID: 19067786] PubMed record related to this compound for laboratory literature review

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Mechanistically, GSH depletion does not cause global protein oxidation but instead selectively oxidizes and destabilizes fatty acid synthase while decreasing lipogenic gene expression at the transcriptional level, collectively suppressing lipogenesis

Notably, when RNF4 was incubated with the CCW16-based PROTAC 2a , we also observed the formation of a covalent bond between 2a and RNF4 at cysteine C91 ( Figure 3D (pink), Supplementary Figure 3C )