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The inhibition of cancer cell growth by GLP-1RAs through regulating cAMP-dependent signaling GLP-1 receptor (GLP-1R) activation leads to adenylyl cyclase activation, subsequently inducing a substantial increase in intracellular cyclic adenosine monophosphate (cAMP) levels
NR4A1 inhibits the ubiquitination degradation of P53 by regulating the downstream target gene MDM2, thereby affecting the oxidative respiration process of mitochondria, producing oxidized lipids, and inducing ferroptosis in cells
doi:10.1515/jpem-2013-0122 90
Learn about cognitive peptides sharing similar neurotrophic mechanisms
Molecular dynamics simulations demonstrated that both MS and MS-CU formed stable complexes with the proteins in a water environment
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