This chronic inflammation, driven by excessive NO signaling, may lead to tissue damage and further facilitate polyp progression, positioning these proteins as promising biomarkers for diagnosis
Houdek MT, Couch CG, Wyles CC, et al
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From Uox knockout mice that establish a human-like uricase-deficient metabolic background, to huXDH and huURAT1 models that support target-specific pharmacology, and huNLRP3/huIL1B models that enable inflammatory pathway evaluation, Cyagens model portfolio is designed to support multiple stages of preclinical research
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