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361 Increased MAM contacts facilitate the initiation and transmission of the ER stress response to reorganize ER and mitochondrial networks
Furthermore, a ferroptosis model of NSCs using erastin treatment was established in vitro, and metformin treatment could reverse the changes in the expression of key ferroptosis-related proteins, increase glutathione synthesis, reduce reactive oxygen species production and improve mitochondrial membrane potential and morphology
The most notable changes include increased expression of Warfarin, LysoPE 22:5, Formiminoglutamic, 2,5-Dimethoxy-4-isopropylthiophenethylamine, and 4-hydroxy-2H-chromen-2-one, along with decreased expression of 1-(beta-D-Ribofuranosyl)-1,4-dihydronicotinamide, Daidzein, Apigenin, Enterolactone, Glu-Leu, Ethoxyquin, and LysoPS 20:5
Lopez-Martinez MI, Miguel M, Garces-Rimon M
When impaired, ERAD is associated with the development of various diseases, such as cancer, metabolic and genetic diseases, and neurodegenerative disorders (Fig
