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It involves ROS accumulation, peroxidation of PUFA-containing phospholipids, and mitochondrial shrinkage [23]
This may indicate extravasation of sclerosing solution either during injection or with endothelial destruction
Autophagy impairment with lysosomal and mitochondrial dysfunction is an important characteristic of oxidative stress-induced senescence
Association of oxidative stress with psychiatric disorders
Significant targets and the effects of PKGI in SMC are activation of myosin light chain phosphatase, inhibition of PLC, inhibition of the IP 3 receptor, inhibition of plasma membrane localized L-type Ca 2+ channels, and activation of plasma membrane localized calcium-activated K + channels (commonly called the big potassium, BK, channels)
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