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We hypothesize that the risk of hyperuricemia in the context of excessive alcohol consumption is multifactorial and related to multiple health outcomes which may have implications for clinical practice

Under physiological conditions, the levels of ROS production are low and can be cleared by the endogenous antioxidant system

Ex-4 also promotes brain-derived insulin production and enhances insulin signaling through the Wnt/-catenin/NeuroD1 signaling pathway, which subsequently reduces tau hyperphosphorylation and cognitive dysfunction in diabetic mice [64]

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