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Recent advances in our understanding of the neuroimmunology of PMS, including the mechanisms that drive slowly expanding lesions, have fuelled optimism for improved treatment of this condition
The distinction separating theoretical risk from observed harm is significant: BPC-157 promotes physiological angiogenesis in the context of tissue injury repair rather than pathological angiogenesis associated with tumor microenvironments
Neither is automatically better
Koroleva SV, Myasoedov NF (2018) Semax as a universal drug for therapy and research
BPC-157 does not cause traditional tolerance or require post-cycle therapy like hormonal compounds
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