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For tumor cells deficient in methythioadenosine phosphorylase (MTAP), Met depletion and inhibition of the key enzyme MAT2A in the Met metabolism pathway are possible therapeutic strategies
doi: 10.2215/CJN.11491116 9 CervantesCEHanounehMJaarBG
A negative value for the valid object count signifies synergism of the CI inhibitors and 5FU, with the opposite true for per cent propidium iodide
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