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Food and Drug Administration (FDA) for safety, efficacy, or any therapeutic purpose
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BMSC-Exos GATA4 inhibited ferroptosis through the upregulation of miR-330-3p, which negatively regulates BAP1, thereby preventing the opening of the mitochondrial permeability transition pore and subsequent mitochondrial dysfunction
[7] Theres also interest in the possibility that the peptide directly improves vascular health, but thats still an emerging scientific field of inquiry
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