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Experimental studies suggest that AST may enhance autophagy and proteasome function, thereby facilitating the clearance of misfolded proteins and limiting cellular stress
Currently, three autophagic mechanisms have been identified: macroautophagy, microautophagy, and chaperone-mediated autophagy (CMA)
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Receptors for GIP are expressed in osteoblasts and bone marrow cells [47]
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