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Hormonal changes, stress, dietary shifts, or new products introduced around the same time as GHK-CU could be responsible
This is a high dose and was used in a research setting
In line with the idea that peripheral immune cells are not passive responders but function as key hubs linking systemic inflammation to central neurodegeneration, we propose that peripheral effects should be considered as mechanistic contributors to neuronal death in PD, as clinically accessible targets for biomarker development, and as potential therapeutic strategies to slow and hopefully halt its progression
Unlike taking growth hormone directly, they do not shut down endogenous production
Clinical studies are primarily investigating Cagrilintide + GLP-1 agonists (like semaglutide)
