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Notably, schisandrin maintained intracellular glutathione (GSH) levels and prevented the depletion of caspase-acting factors that glutamate toxicity would normally cause (58)

During IVM, FLI medium activates key signaling pathways, including the phosphorylation of protein kinase B (AKT), mitogen-activated protein kinase 1/3 (MAPK1/3), signal transducer and activator of transcription 3 (STAT3), and the mammalian target of rapamycin (mTOR) downstream target ribosomal protein S6 kinase B1 (RPS6KB1) (Nahar et al., 2024)

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6.6 Personalized medicine and multi-pathway synergistic targeting The heterogeneity of melanoma necessitates personalized therapeutic approaches tailored to the unique molecular and metabolic landscape of each patients tumor (211, 212)