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In toxin-induced PD mouse models, DA-JC1, a novel dual GLP-1/GIP receptor agonist, improved motor impairment, increased the number of dopaminergic neurons in the substantia nigra, enhanced BDNF levels, and modulated apoptosis signaling pathways by altering the expression of Bax and Bcl-2 [89, 90]

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However, the American Diabetes Association Standards for the Management of Diabetes recommend a combination approach to treat type 2 diabetes, rather than a stepwise escalation of therapy based on the patient's condition (Figure 1)