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Studies have demonstrated that GLP-1 receptor activation may reduce production of pro-inflammatory cytokines such as tumor necrosis factor-alpha (TNF-), interleukin-6 (IL-6), and interleukin-1 beta (IL-1) the same inflammatory mediators that drive joint destruction in rheumatoid arthritis

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Protein kinase C pathway mediates the protective effects of glucagon-like peptide-1 on the apoptosis of islet -cells

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