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Our group recently described that a genetic disruption of the xCT subunit using CRISPR-Cas9 inhibits protein synthesis and proliferation in vitro ( 14 C-cystine transport assay in xCT knockout (xCT-KO) cells revealed this transporter as unique and indispensible for cystine uptake, as a complete abolishment of cystine transport has been observed

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Without enough of it, that process slows down considerably

Complex II inhibition suppresses RSL3-induced ferroptosis by restoring mitochondrial bioenergetics

Vial seal integrity Once vial seal is compromised, shelf life decreases