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The review follows a shift in oversight to potentially increase access to these substances

proposed that exenatide upregulates uncoupling protein-3 (UCP-3) and nuclear respiratory factor-1 (Nrf-1) in cardiomyocytes subjected to ischemia/reperfusion (I/R) via the cAMP/PKA pathway, thereby facilitating mitochondrial homeostasis [93]

The rationale is mechanistic: the two compounds operate through distinct but non-overlapping pathways, with tissue targets that complement rather than duplicate each other

The numbers above come straight from the clinical trials that supported the FDA approval

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