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gossypii causing resistance to a glycine antimetabolite, aminomethylphosphonic acid, yielded improved producers (Sugimoto et al., 2010)

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Intracellular glutamine level rises in PIK3CA -MT HCT116 colorectal cancer cells following aspirin administration and under hypoxic conditions Given the observed link between aspirin treatment and amino acid transporter expression under hypoxia in PIK3CA -mutant cells, we next explored whether these changes are accompanied by alterations in intracellular glutamine metabolism